Idiopathic nephrotic syndrome: prevention of early relapse.
نویسندگان
چکیده
منابع مشابه
Immunopathogenesis of idiopathic nephrotic syndrome.
Idiopathic nephrotic syndrome is the most frequent glomerular disease in children. The mechanisms underlying its pathophysiology have been investigated by genetic, cellular and molecular approaches. While genetic analyses have provided new insights into disease pathogenesis through the discovery of several podocyte genes mutated in distinct forms of inherited nephrotic syndrome, the molecular b...
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Chyloperitoneum associated with nephrotic syndrome is rare clinical entity and characterized by milky ascites. The modalities for detection of chyloperitoneum are laboratory investigations and radiological images. Lipoprotein electrophoresis is useful for confirmation of the diagnosis with chyloperitoneum. Treating underlying disease is the most important in the management of these patients.
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The objective of this study was to identify predictors of relapse and determine the predictive score for relapse in steroid-sensitive nephrotic syndrome. Ninety-nine children with nephrotic syndrome visiting the pediatric nephrology outpatient clinic of Soetomo Hospital from 1983 to 2001 were studied. There were 63 children with relapses (50 infrequent relapses, 13 frequent relapses) and 36 chi...
متن کاملPathophysiology of oedema in idiopathic nephrotic syndrome.
The decrease in plasma protein and colloid osmotic pressure (COP) in the nephrotic syndrome is accompanied by a decrease in tissue-fluid protein and COP. The latter protects against a fall in blood volume. However, the range and speed of this protective mechanism are limited, and a decrease in blood volume can be expected if plasma COP is below approximately 10 mmHg, or (temporarily) if the pro...
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A controlled trial of disodium cromoglycate treatment of steroid-responsive nephrotic syndrome failed to show a therapeutic effect on the tendency to relapse after withdrawal of corticosteroids.
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ژورنال
عنوان ژورنال: BMJ
سال: 1978
ISSN: 0959-8138,1468-5833
DOI: 10.1136/bmj.1.6117.892